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normal retinal astrocytes  (Innoprot Inc)


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    Innoprot Inc normal retinal astrocytes
    Cultures of normal human <t>astrocytes</t> respond to vitreous exposure. RELN, Aβ1-42, FTH1, TAU and GFAP transcript expressions by ERM vitreal fluid exposed astrocytes. The lines represent the exclusivity of expression for: ( A ) red line for stage 3 and green line for stage 2 and ( B ) red line for stage 4 and green line for stage 2; p < 0.001, REST–ANOVA and multiparametric analysis with Bonferroni correction. Legend: RELN—Reelin; Aβ1-42—amyloid β 1-42; FTH1—ferritin heavy chain; TAU—TAU protein; GFAP—glial fibrillary acidic protein.
    Normal Retinal Astrocytes, supplied by Innoprot Inc, used in various techniques. Bioz Stars score: 94/100, based on 4 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/normal+retinal+astrocytes/pmc12384615-113-8-11?v=Innoprot+Inc
    Average 94 stars, based on 4 article reviews
    normal retinal astrocytes - by Bioz Stars, 2026-07
    94/100 stars

    Images

    1) Product Images from "Expression of Reelin, Aβ1-42, Tau and FTH1 in Idiopathic Epiretinal Membranes: Exploring the Link Between Reelin and Neurodegenerative Biomarkers"

    Article Title: Expression of Reelin, Aβ1-42, Tau and FTH1 in Idiopathic Epiretinal Membranes: Exploring the Link Between Reelin and Neurodegenerative Biomarkers

    Journal: Biomolecules

    doi: 10.3390/biom15081187

    Cultures of normal human astrocytes respond to vitreous exposure. RELN, Aβ1-42, FTH1, TAU and GFAP transcript expressions by ERM vitreal fluid exposed astrocytes. The lines represent the exclusivity of expression for: ( A ) red line for stage 3 and green line for stage 2 and ( B ) red line for stage 4 and green line for stage 2; p < 0.001, REST–ANOVA and multiparametric analysis with Bonferroni correction. Legend: RELN—Reelin; Aβ1-42—amyloid β 1-42; FTH1—ferritin heavy chain; TAU—TAU protein; GFAP—glial fibrillary acidic protein.
    Figure Legend Snippet: Cultures of normal human astrocytes respond to vitreous exposure. RELN, Aβ1-42, FTH1, TAU and GFAP transcript expressions by ERM vitreal fluid exposed astrocytes. The lines represent the exclusivity of expression for: ( A ) red line for stage 3 and green line for stage 2 and ( B ) red line for stage 4 and green line for stage 2; p < 0.001, REST–ANOVA and multiparametric analysis with Bonferroni correction. Legend: RELN—Reelin; Aβ1-42—amyloid β 1-42; FTH1—ferritin heavy chain; TAU—TAU protein; GFAP—glial fibrillary acidic protein.

    Techniques Used: Expressing

    Graphical illustration of inflammation loop during Alzheimer’s disease. A representative image showing how brain and eye (retina) are involved in Alzheimer’s disease (a neurodegenerative disease) and some ocular conditions (vitreoretinal traction and AMD). As stated, β-amyloid accumulation (mainly Aβ1-42 inside soluble and insoluble plaques) and neurofibrillary tangles (chiefly the hyperphosphorylated form) result in a substantial synaptic and neuronal loss . In AD brains, activated microglia and reactive astrocytes are associated with plaques and tangles, so their activation in retinal tissues might be of great relevance for understanding the neurodegenerative microenvironment . The concept behind “burning person” is that the mechanisms of inflammation and oxidative stress that occur in the brain may have similarities or effects in the retina. Legend: Aβ—beta-amyloid; TAU—microtubule-associated protein TAU (tangles); ILs—interleukins; Fe—iron; ROS—reactive oxygen species indicative of oxidative stress; AMD—age-related macular degeneration; NU—neutrophil; M—microglia; AS—astrocyte; NU—neuron; AD—Alzheimer’s disease.
    Figure Legend Snippet: Graphical illustration of inflammation loop during Alzheimer’s disease. A representative image showing how brain and eye (retina) are involved in Alzheimer’s disease (a neurodegenerative disease) and some ocular conditions (vitreoretinal traction and AMD). As stated, β-amyloid accumulation (mainly Aβ1-42 inside soluble and insoluble plaques) and neurofibrillary tangles (chiefly the hyperphosphorylated form) result in a substantial synaptic and neuronal loss . In AD brains, activated microglia and reactive astrocytes are associated with plaques and tangles, so their activation in retinal tissues might be of great relevance for understanding the neurodegenerative microenvironment . The concept behind “burning person” is that the mechanisms of inflammation and oxidative stress that occur in the brain may have similarities or effects in the retina. Legend: Aβ—beta-amyloid; TAU—microtubule-associated protein TAU (tangles); ILs—interleukins; Fe—iron; ROS—reactive oxygen species indicative of oxidative stress; AMD—age-related macular degeneration; NU—neutrophil; M—microglia; AS—astrocyte; NU—neuron; AD—Alzheimer’s disease.

    Techniques Used: Activation Assay



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    normal retinal astrocytes  (Innoprot Inc)
    94
    Innoprot Inc normal retinal astrocytes
    Cultures of normal human <t>astrocytes</t> respond to vitreous exposure. RELN, Aβ1-42, FTH1, TAU and GFAP transcript expressions by ERM vitreal fluid exposed astrocytes. The lines represent the exclusivity of expression for: ( A ) red line for stage 3 and green line for stage 2 and ( B ) red line for stage 4 and green line for stage 2; p < 0.001, REST–ANOVA and multiparametric analysis with Bonferroni correction. Legend: RELN—Reelin; Aβ1-42—amyloid β 1-42; FTH1—ferritin heavy chain; TAU—TAU protein; GFAP—glial fibrillary acidic protein.
    Normal Retinal Astrocytes, supplied by Innoprot Inc, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/normal+retinal+astrocytes/pmc12384615-113-8-11?v=Innoprot+Inc
    Average 94 stars, based on 1 article reviews
    normal retinal astrocytes - by Bioz Stars, 2026-07
    94/100 stars
    		  Buy from Supplier

    Image Search Results


    Cultures of normal human astrocytes respond to vitreous exposure. RELN, Aβ1-42, FTH1, TAU and GFAP transcript expressions by ERM vitreal fluid exposed astrocytes. The lines represent the exclusivity of expression for: ( A ) red line for stage 3 and green line for stage 2 and ( B ) red line for stage 4 and green line for stage 2; p < 0.001, REST–ANOVA and multiparametric analysis with Bonferroni correction. Legend: RELN—Reelin; Aβ1-42—amyloid β 1-42; FTH1—ferritin heavy chain; TAU—TAU protein; GFAP—glial fibrillary acidic protein.

    Journal: Biomolecules

    Article Title: Expression of Reelin, Aβ1-42, Tau and FTH1 in Idiopathic Epiretinal Membranes: Exploring the Link Between Reelin and Neurodegenerative Biomarkers

    doi: 10.3390/biom15081187

    Figure Lengend Snippet: Cultures of normal human astrocytes respond to vitreous exposure. RELN, Aβ1-42, FTH1, TAU and GFAP transcript expressions by ERM vitreal fluid exposed astrocytes. The lines represent the exclusivity of expression for: ( A ) red line for stage 3 and green line for stage 2 and ( B ) red line for stage 4 and green line for stage 2; p < 0.001, REST–ANOVA and multiparametric analysis with Bonferroni correction. Legend: RELN—Reelin; Aβ1-42—amyloid β 1-42; FTH1—ferritin heavy chain; TAU—TAU protein; GFAP—glial fibrillary acidic protein.

    Article Snippet: We developed in vitro primary cultures of human normal retinal astrocytes (Innoprot; Bizkaia, Spain).

    Techniques: Expressing

    Graphical illustration of inflammation loop during Alzheimer’s disease. A representative image showing how brain and eye (retina) are involved in Alzheimer’s disease (a neurodegenerative disease) and some ocular conditions (vitreoretinal traction and AMD). As stated, β-amyloid accumulation (mainly Aβ1-42 inside soluble and insoluble plaques) and neurofibrillary tangles (chiefly the hyperphosphorylated form) result in a substantial synaptic and neuronal loss . In AD brains, activated microglia and reactive astrocytes are associated with plaques and tangles, so their activation in retinal tissues might be of great relevance for understanding the neurodegenerative microenvironment . The concept behind “burning person” is that the mechanisms of inflammation and oxidative stress that occur in the brain may have similarities or effects in the retina. Legend: Aβ—beta-amyloid; TAU—microtubule-associated protein TAU (tangles); ILs—interleukins; Fe—iron; ROS—reactive oxygen species indicative of oxidative stress; AMD—age-related macular degeneration; NU—neutrophil; M—microglia; AS—astrocyte; NU—neuron; AD—Alzheimer’s disease.

    Journal: Biomolecules

    Article Title: Expression of Reelin, Aβ1-42, Tau and FTH1 in Idiopathic Epiretinal Membranes: Exploring the Link Between Reelin and Neurodegenerative Biomarkers

    doi: 10.3390/biom15081187

    Figure Lengend Snippet: Graphical illustration of inflammation loop during Alzheimer’s disease. A representative image showing how brain and eye (retina) are involved in Alzheimer’s disease (a neurodegenerative disease) and some ocular conditions (vitreoretinal traction and AMD). As stated, β-amyloid accumulation (mainly Aβ1-42 inside soluble and insoluble plaques) and neurofibrillary tangles (chiefly the hyperphosphorylated form) result in a substantial synaptic and neuronal loss . In AD brains, activated microglia and reactive astrocytes are associated with plaques and tangles, so their activation in retinal tissues might be of great relevance for understanding the neurodegenerative microenvironment . The concept behind “burning person” is that the mechanisms of inflammation and oxidative stress that occur in the brain may have similarities or effects in the retina. Legend: Aβ—beta-amyloid; TAU—microtubule-associated protein TAU (tangles); ILs—interleukins; Fe—iron; ROS—reactive oxygen species indicative of oxidative stress; AMD—age-related macular degeneration; NU—neutrophil; M—microglia; AS—astrocyte; NU—neuron; AD—Alzheimer’s disease.

    Article Snippet: We developed in vitro primary cultures of human normal retinal astrocytes (Innoprot; Bizkaia, Spain).

    Techniques: Activation Assay